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Omega-3 vetzuren verminderen kans op darmkanker flink.*

Uit een onderzoek onder 460 mensen waarvan 178 mannen met darmkanker blijkt dat zij met de de hoogste bloedwaarde aan omega-3 vetzuur wel 66 % minder kans hebben op het krijgen van darmkanker. Omega-3 vetzuren remmen het COX-2 enzym en daardoor ontstekingsreacties met de goede gevolgen. Aspirine doet hetzelfde als omega-3 vetzuren met het COX-2 enzym, vandaar dat gebruikers van aspirine in deze studie geen extra voordeel hadden. Omega-3 vetzuren zitten vooral in vette vis  en lijnzaad.

Blood Levels of Long-Chain Polyunsaturated Fatty Acids, Aspirin, and the Risk of Colorectal Cancer

Megan N. Hall1,2, Hannia Campos1, Haojie Li3, Howard D. Sesso4, Meir J. Stampfer1,2,3,4, Walter C. Willett1,2,3 and Jing Ma3

Departments of 1 Nutrition and 2 Epidemiology, Harvard School of Public Health; 3 Channing Laboratory and 4 Division of Preventive Medicine, Department of Medicine, Harvard Medical School and Brigham and Women's Hospital, Boston, Massachusetts

Requests for reprints: Jing Ma, Channing Laboratory, Department of Medicine, Brigham and Women's Hospital and Room 336, Harvard Medical School, 181 Longwood Ave, Boston, MA 02115. Phone: 617-525-2708; Fax: 617-525-2008. E-mail: jing.ma@channing.harvard.edu

Background: N-3 fatty acids may decrease risk of colorectal cancer by inhibiting the cyclooxygenase-2 enzyme and production of proinflammatory eicosanoids derived from arachidonic acid (20:4n-6). Aspirin also inhibits the cyclooxygenase-2 enzyme and may share with n-3 fatty acids a potential mechanism to decrease the risk of colorectal cancer.

Methods: We conducted a nested case-control analysis using blood samples collected from the Physicians' Health Study participants in 1982 to 1984. N-3 and n-6 fatty acid levels were measured using gas-liquid chromatography for 178 men who developed colorectal cancer through December 31, 1995 and 282 age- and smoking-matched controls. We used conditional logistic regression to examine associations. All statistical tests were two-sided.

Results: Total long-chain n-3 fatty acids were nonsignificantly inversely associated with colorectal cancer risk [relative risk (RR) for highest versus lowest quartile, 0.60; 95% confidence interval (95% CI), 0.32 to 1.11; Ptrend = 0.10], after adjustment for possible confounders. We observed potential interaction between randomized aspirin assignment and long-chain n-3 fatty acid levels (Pinteraction = 0.04). Among men not on aspirin, RRs (95% CI) for increasing quartiles of long-chain n-3 fatty acids were 1.00 (reference), 0.60 (0.28-1.28), 0.51 (0.22-1.17), and 0.34 (0.15-0.82), Ptrend = 0.006. For participants taking aspirin, there was no additional benefit of increasing n-3 fatty acid levels. The RR (95% CI) for the highest versus lowest quartile of n-6 fatty acids was 0.64 (0.35-1.17).

Conclusions: Blood levels of long-chain n-3 fatty acids were associated with decreased risk of colorectal cancer among men not using aspirin. N-6 fatty acids were nonsignificantly inversely associated with colorectal cancer risk. (Cancer Epidemiol Biomarkers Prev 2007;16(2):314–21) (Maart 2007)  (Opm. Meer over voeding en vetzuren)

 

 

 

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